• What Happened to Mad Cow Disease?

    If you were around in the '90s, you might remember the scare over mad cow disease, but it seems to have quieted down in the intervening years. What happened? We're conducting a survey of our viewers! If you have time, please give us feedback: https://www.surveymonkey.com/r/SciShowSurvey2017 Hosted by: Stefan Chin ---------- Support SciShow by becoming a patron on Patreon: https://www.patreon.com/scishow ---------- Dooblydoo thanks go to the following Patreon supporters: D.A. Noe, Nicholas Smith, سلطان الخليفي, Piya Shedden, KatieMarie Magnone, Scott Satovsky Jr, Bella Nash, Charles Southerland, Patrick D. Ashmore, Tim Curwick, charles george, Kevin Bealer, Philippe von Bergen, Chris Peters, Fatima Iqbal ---------- Looking for SciShow elsewhere on the internet? Facebook: http://www.facebo...

    published: 20 Sep 2017
  • Report on UK variant CJD in a second genetic type (2008)

    Click herehttp://www.foodsafetypolicy.com/comment-variant-cjd-in-second-genetic-type to find out more.

    published: 02 Jul 2010
  • American Report on BSE in Europe (2000)

    Click herehttp://www.foodsafetypolicy.com

    published: 30 Dec 2008
  • Human 'Mad Cow Disease' (CJD) News Report No. 2

    Click herehttp://www.foodsafetypolicy.com On December 2007, at just 24 years of age, Andy Black died after suffering from the human form of mad cow's disease vCJD (variant Creutzfeldt-Jakob Disease). Andy worked in the media producing/researching programmes for talkSPORT Radio, BBC, and ITV. During his terrible last days and at his request Christine Lord made it her mission to find out "Who killed my son?" and with the BBC, she produced a documentary exposing the key players at the centre of the BSE crisis whose actions and decisions led to his untimely and avoidable death. The Documentary is at http://www.youtube.com/watch?v=MAJTr6Nxxa0 Christine Lord, as Andy's mum and a freelance journalist continues to research the scandal - and its links between school meals, (state and indepen...

    published: 01 Oct 2013
  • BSE symptom video

    published: 02 May 2015
  • BSE and CJD in the U.K. explained

    Click here http://www.foodsafetypolicy.com/bseandcjdexplained Bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, is a fatal neurodegenerative disease in cattle that causes a spongy degeneration in the brain and spinal cord. BSE has a long incubation period, about 30 months to 8 years, usually affecting adult cattle at a peak age onset of four to five years, all breeds being equally susceptible. In the United Kingdom, the country worst affected, more than 180,000 cattle have been infected and 4.4 million slaughtered during the eradication program. The disease may be most easily transmitted to human beings by eating food contaminated with the brain, spinal cord or digestive tract of infected carcasses. However, it should also be noted that the infectious agent, al...

    published: 06 Aug 2010
  • Human BSE and compensation for CJD families

    Click herehttp://www.foodsafetypolicy.com

    published: 04 Dec 2008
  • Theory on the origins of BSE (2005): United Kingdom video

    Click herehttp://www.foodsafetypolicy.com New variant CJD may have come from human remains which were put into cattle feed. This theory has been criticised but it is some interesting research into the causes of BSE. *** CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts. The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordinati...

    published: 30 Dec 2008
  • Human 'Mad Cow Disease' (CJD) News Report

    Click herehttp://www.foodsafetypolicy.com *** On 16th December 2007, at just 24 years of age, Andy Black died after suffering from the human form of mad cow's disease vCJD (variant Creutzfeldt-Jakob Disease). Andy worked in the media producing/researching programmes for talkSPORT Radio, BBC, and ITV. During his terrible last days and at his request Christine Lord made it her mission to find out "Who killed my son?" and with the BBC, she produced a documentary exposing the key players at the centre of the BSE crisis whose actions and decisions led to his untimely and avoidable death. The Documentary is at http://www.youtube.com/watch?v=MAJTr6Nxxa0 Christine Lord, as Andy's mum and a freelance journalist continues to research the scandal - and its links between school meals, (state...

    published: 29 Nov 2008
  • Detecting Prions and Diagnosing Prion Diseases by Byron Caughey

    "Progress in Detecting Prions and Diagnosing Prion Diseases" is lectured at the CJD foundation 2015 conference by Byron Caughey. Discussing Creutzfeldt-Jakob Disease (CJD) and other Prion diseases, Bryon Caughey helps shed some light on these health issues. Byron Caughey is from the Laboratory of Persistent Viral Diseases, NIH/NIAID Rocky Mountain Laboratories, Hamilton, Montana. Caughey discusses the progress being made to diagnose various protein misfolding diseases in living humans. Topics discussed include: prion diseases, transmissible spongiform encephalopathies, chronic wasting disease, neurodegenerative diseases, bank voles, sporadic CJD,familial CJD, scrapie, GSS, fatal familial insomnia, iatrogenic CJD,. dura mater transplant, growth hormone injections, kuru in New Guinea, pro...

    published: 25 Aug 2015
  • BSE - Clinical Signs - Sensory Disturbances

    published: 03 Dec 2014
  • Bovine spongiform encephalopathy

    Bovine spongiform encephalopathy (BSE) is characterized by behavioural changes, locomotory disturbaances and loss of condition. The course of the disease is about 1-3 months terminating in death in some animals, but many are slaughtered because of unmanageable behaviour or trauma due to failling. Affected cattle were first identified in the United Kingdom in 1986, as a result of feeding contaminated ruminant protein and affected nearly 200 000 cattle. Since then BSE has been reported in several other countries. Variant Creutzfeldt-Jacob disease in humans most likely results from the consumption of beef products contaminated by central nervous system tissue. The complete learning module is available through the African Veterinary Information Portal (AfriVIP) at: http://www.afrivip.org...

    published: 20 Feb 2013
  • BSE - UK

    July 2005 The cost of the crisis over BSE, and its links to the human variant CJD were obvious to all in terms of cattle and livelihood. However, the question of the role that cost had to play before the crisis erupted is one that is much more contentious. Ron McCullagh illustrates both the devastating impact of the crisis, and the debates that still rage over the nature and treatment of the disease.

    published: 07 Nov 2007
  • Hyperekplexia as the presenting symptom of Creutzfeldt-Jakob disease

    We describe a patient with hyperekplexia as the initial presenting symptom in sporadic Creutzfeldt-Jakob disease (CJD). We also demonstrate the utility of the sensitive and highly specific new prion protein amplification assay, real-time quaking-induced conversion (RT-QUIC), which amplifies and detects misfolded β-sheet–rich conformers of the prion protein (PrPsc), in expediting a confident clinical diagnosis of CJD in an atypical case. For more, see: http://cp.neurology.org/content/5/6/498.extract

    published: 09 Dec 2015
  • Creutzfeldt-Jakob disease (CJD)

    Video of a patient suffering from Creutzfeldt-Jakob disease (CJD). For Doctors - Visit http://doctorshangout.com/video/video/show?id=2002836%3AVideo%3A92991 For Patients - Join http://www.patient.org.in - It is a health based social network of people sharing their disease experiences, treatment options, knowledge and giving support to other people suffering from similar health problems.

    published: 21 Aug 2008
  • Part 1: Mad Cow Disease and Food Safety News Programme

    Click herehttp://www.foodsafetypolicy.com/documentary BBC Documentary about Andy Black (from Portsmouth, England) who died after suffering from the human form of mad cow disease variant CJD. vCJd fact file Creutzfeldt-Jakob disease is a rare and fatal progressive degenerative brain disease. It is one of a group of diseases which affect humans and animals called Transmissible Spongiform Encephalopathies (TSEs). TSEs are believed to be caused by the build up of an abnormal form of the naturally occurring 'prion' protein in the brain. CJD was originally described in its classical form in 1920. A new variant - called variant CJD (vCJD) - was first identified in 1996. Variant CJD is strongly linked to exposure to a TSE of cattle called Bovine Spongiform Encephalopathy (BSE), probably th...

    published: 15 Dec 2008
  • Ministerial Statements on BSE and CJD: 1990 - 2000

    Click herehttp://www.foodsafetypolicy.com Ministerial Statements on U.K. BSE and CJD: 1990 - 2000 1) Agriculture Minister from 1990 2) Prime Minister from 1995 3) Health Minister from 1996 4) Agriculture Minister from 2000

    published: 16 Aug 2010
  • What causes BSE, CJD & MS? Organic Farmer Mark Purdey on Organophosphates (2001)

    Mark Purdey and Organophosphate (Film/Video, i-Contact, September 2001) Organic livestock farmer Mark Purdey was ordered in the 1980's, along with all cattle and dairy farmers in the UK, to treat his cows with an organophosphate pesticide 'Phosmet' manufactured by I.C.I.. Organophosphates are derived from military nerve gas and a systemic treatment (the chemical enters the entire internal system of the cow) would undermine his organic principles so Mark refused to treat. "If Phosmet is proven to have caused BSE, the worldwide use of organophosphates (OPs) could be put into jeopardy, costing the chemical industry billions. The government know more than they're letting on. They've stuck to the scrapie theory to placate people and give the impression they've got it under control." Mark P...

    published: 16 Mar 2011
  • Creutzfeldt-Jakob Disease Report from 2010

    See http://www.foodsafetypolicy.com CJD Research Campaign from October 2011 Two decades on since BSE or mad cow disease hit the headlines, a father of a victim of its form in humans, new variant CJD, wants the government to maintain research into the degenerative condition. Recent research suggests up to 1 in 1000 people could still be affected. *** Creutzfeldt--Jakob disease or CJD is a degenerative neurological disorder (brain disease) that is incurable and invariably fatal.CJD is at times called a human form of mad cow disease, given that bovine spongiform encephalopathy is believed to be the cause of variant Creutzfeldt--Jakob disease in humans. CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops hol...

    published: 12 Sep 2011
  • Mad Cow Disease and Food Safety News Programme (Full length)

    See http://www.foodsafetypolicy.com/documentary for the interview towards the end of the programme. Joe Crowley's (the presenter's) filming diary... For me and friends of my age, Mad Cow Disease seems like a distant nightmare - something that haunted us on the news as we grew up. But this Inside Out programme has made me think again and, sadly, given the disease a painful new relevance. It's about a young guy like me, starting out on a career in the media, when he was struck down by the human form of the disease. And it's a story of his mother's (Christine Lord) relentless search for answers, as she tries to find out who killed her son (Andrew Black). *** Further informarion on variant CJD Inside Out looks at the rise of Variant CJD, the symptoms and the support available for thos...

    published: 01 Jul 2011
  • What is Mad Cow Disease?

    Mad Cow Disease - What to Know - as part of the education series by GeoBeats. Mad Cow Disease - What is It? Scientifically named Bovine Spongiform Encephalopathy, it is a progressive neurological disease that affects a cow's nervous system. Once infected, the cow's behavior becomes erratic - inability to walk, uncontrolled movements and lower milk production. Incubation period can be months, even years. There's no known treatment and the disease is fatal. How Do Cows Get Infected? A cow gets infected with the disease if they eat diseased feed which has abnormal proteins called prions found in brains, spinal cords, and other parts. These prions cause the degeneration in brain, giving it a spongy like appearance. How Can It Affect Us? Humans don't get mad cow disease as such but they...

    published: 30 Apr 2012
  • UK: TEENAGER WHO DIED FROM HUMAN EQUIVALENT OF MAD COW DISEASE

    English/Nat The parents of a teenager who died from the human equivalent of Mad Cow Disease hit out Thursday at the British government's handling of the issue. Stephen Churchill was 19 when he died last year from Disease. The government has only now admitted there's a link between the two conditions. Their grief dates from a year ago - but their anger is new. Dorothy and David Churchill say the death of their son Stephen had been a mystery - until now. They recall how his illness started. SOUNDBITE: (English) "All the things that you and I do naturally, take for granted, Stephen couldn't do. He couldn't feed himself, he couldn't walk, he had no coordination, his short term memory went, couldn't write and he actually was only in bed the last few days of his life...

    published: 21 Jul 2015
  • Creutzfeldt-Jakob Disease (CJD) "Mad Cow Disease"- My Father's Story (with captions english-espanol)

    Rafael Alvaro Bohorquez Villamil passed away after a brave 9 weeks from a very rare disease: Creutzfeldt-Jakob Disease (CJD) on October 28, 2013, after only a few months of arriving to Canada as a tourist where he was enjoying his life and good health with his youngest grandson. Everyone he met in Toronto in this short period saw what a great man he was, warm and joyful, full of peace. Everyone that knew him in other parts of the world already knew this about him.Loving father,grandfather, brother, uncle and boss and friend, he will be greatly missed and will remain forever in out hearts. Any donations to help pay hospital bills will be appreciated and cheques can be written directly to the hospital: North York General Hospital BOHORQUEZ VILLAMIL MRN1735438 Or if you would like to use GO ...

    published: 13 Nov 2013
  • Mad Cow Disease-vCJD

    published: 11 Apr 2016
developed with YouTube
What Happened to Mad Cow Disease?
4:32

What Happened to Mad Cow Disease?

  • Order:
  • Duration: 4:32
  • Updated: 20 Sep 2017
  • views: 460056
videos
If you were around in the '90s, you might remember the scare over mad cow disease, but it seems to have quieted down in the intervening years. What happened? We're conducting a survey of our viewers! If you have time, please give us feedback: https://www.surveymonkey.com/r/SciShowSurvey2017 Hosted by: Stefan Chin ---------- Support SciShow by becoming a patron on Patreon: https://www.patreon.com/scishow ---------- Dooblydoo thanks go to the following Patreon supporters: D.A. Noe, Nicholas Smith, سلطان الخليفي, Piya Shedden, KatieMarie Magnone, Scott Satovsky Jr, Bella Nash, Charles Southerland, Patrick D. Ashmore, Tim Curwick, charles george, Kevin Bealer, Philippe von Bergen, Chris Peters, Fatima Iqbal ---------- Looking for SciShow elsewhere on the internet? Facebook: http://www.facebook.com/scishow Twitter: http://www.twitter.com/scishow Tumblr: http://scishow.tumblr.com Instagram: http://instagram.com/thescishow ---------- Sources: http://www.oie.int/en/animal-health-in-the-world/bse-specific-data/number-of-cases-in-the-united-kingdom/ https://www.cdc.gov/prions/bse/about.html http://www.bseinfo.org/bseclinicalsignssymptoms.aspx https://www.scientificamerican.com/article/what-is-a-prion-specifica/ http://www.hopkinsmedicine.org/healthlibrary/conditions/nervous_system_disorders/prion_diseases_134,56/ http://www.mayoclinic.org/diseases-conditions/creutzfeldt-jakob-disease/basics/definition/con-20028005 https://www.fda.gov/BiologicsBloodVaccines/BloodBloodProducts/QuestionsaboutBlood/DonatingBlood/ http://www.who.int/mediacentre/factsheets/fs180/en/ https://www.cdc.gov/prions/vcjd/vcjd-reported.html https://www.cdc.gov/prions/vcjd/risk-travelers.html ---------- Images: https://en.wikipedia.org/wiki/File:Aphis.usda.gov_BSE_5.jpg
https://wn.com/What_Happened_To_Mad_Cow_Disease
Report on UK variant CJD in a second genetic type (2008)
6:40

Report on UK variant CJD in a second genetic type (2008)

  • Order:
  • Duration: 6:40
  • Updated: 02 Jul 2010
  • views: 692
videos
Click herehttp://www.foodsafetypolicy.com/comment-variant-cjd-in-second-genetic-type to find out more.
https://wn.com/Report_On_UK_Variant_Cjd_In_A_Second_Genetic_Type_(2008)
American Report on BSE in Europe (2000)
8:06

American Report on BSE in Europe (2000)

  • Order:
  • Duration: 8:06
  • Updated: 30 Dec 2008
  • views: 567
videos
Click herehttp://www.foodsafetypolicy.com
https://wn.com/American_Report_On_Bse_In_Europe_(2000)
Human 'Mad Cow Disease' (CJD) News Report No. 2
3:28

Human 'Mad Cow Disease' (CJD) News Report No. 2

  • Order:
  • Duration: 3:28
  • Updated: 01 Oct 2013
  • views: 21947
videos
Click herehttp://www.foodsafetypolicy.com On December 2007, at just 24 years of age, Andy Black died after suffering from the human form of mad cow's disease vCJD (variant Creutzfeldt-Jakob Disease). Andy worked in the media producing/researching programmes for talkSPORT Radio, BBC, and ITV. During his terrible last days and at his request Christine Lord made it her mission to find out "Who killed my son?" and with the BBC, she produced a documentary exposing the key players at the centre of the BSE crisis whose actions and decisions led to his untimely and avoidable death. The Documentary is at http://www.youtube.com/watch?v=MAJTr6Nxxa0 Christine Lord, as Andy's mum and a freelance journalist continues to research the scandal - and its links between school meals, (state and independent), baby food, vaccines and medicines. *** Creutzfeldt--Jakob disease or CJD is a degenerative neurological disorder (brain disease) that is incurable and invariably fatal. CJD is at times called a human form of mad cow disease, given that bovine spongiform encephalopathy is believed to be the cause of variant Creutzfeldt--Jakob disease in humans. CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts. The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordination dysfunction (ataxia), changes in gait, rigid posture, and seizures. The duration of the disease varies greatly, but sporadic (non-inherited) CJD can be fatal within months or even weeks (Johnson, 1998). In some people, the symptoms can continue for years. In most patients, these symptoms are followed by involuntary movements and the appearance of an atypical diagnostic electroencephalogram tracing. Most victims die 6 months after initial symptoms appear, often of pneumonia due to impaired coughing reflexes. About 15% of patients survive 2 or more years. The symptoms of CJD are caused by the progressive death of the brain's nerve cells, which is associated with the build-up of abnormal prion proteins forming amyloids. When brain tissue from a CJD patient is examined under a microscope, many tiny holes can be seen where whole areas of nerve cells have died. The word "spongiform" in "transmissible spongiform encephalopathies" refers to the sponge-like appearance of the brain tissue.
https://wn.com/Human_'Mad_Cow_Disease'_(Cjd)_News_Report_No._2
BSE symptom video
0:48

BSE symptom video

  • Order:
  • Duration: 0:48
  • Updated: 02 May 2015
  • views: 53305
videos
https://wn.com/Bse_Symptom_Video
BSE and CJD in the U.K. explained
1:18

BSE and CJD in the U.K. explained

  • Order:
  • Duration: 1:18
  • Updated: 06 Aug 2010
  • views: 1274
videos
Click here http://www.foodsafetypolicy.com/bseandcjdexplained Bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, is a fatal neurodegenerative disease in cattle that causes a spongy degeneration in the brain and spinal cord. BSE has a long incubation period, about 30 months to 8 years, usually affecting adult cattle at a peak age onset of four to five years, all breeds being equally susceptible. In the United Kingdom, the country worst affected, more than 180,000 cattle have been infected and 4.4 million slaughtered during the eradication program. The disease may be most easily transmitted to human beings by eating food contaminated with the brain, spinal cord or digestive tract of infected carcasses. However, it should also be noted that the infectious agent, although most highly concentrated in nervous tissue, can be found in virtually all tissues throughout the body, including blood. In humans, it is known as new variant Creutzfeldt--Jakob disease (vCJD or nvCJD), and by October 2009, it had killed 166 people in the United Kingdom, and 44 elsewhere[5] Between 460,000 and 482,000 BSE-infected animals had entered the human food chain before controls on high-risk offal were introduced in 1989. A British inquiry into BSE concluded that the epizootic was caused by cattle, who are normally herbivores, being fed the remains of other cattle in the form of meat and bone meal (MBM), which caused the infectious agent to spread. There are studies indicating that the cause of BSE may be from the contamination of MBM from sheep with scrapie that were processed in the same slaughterhouse. The epidemic was probably accelerated by the recycling of infected bovine tissues prior to the recognition of BSE. The origin of the disease itself remains unknown. The infectious agent is distinctive for the high temperatures at which it remains viable; this contributed to the spread of the disease in the United Kingdom, which had reduced the temperatures used during its rendering process. Another contributory factor was the feeding of infected protein supplements to very young calves. This first reported case in North America was in December 1993 from Alberta, Canada. Another case reported later in May 2003. The first known U.S. occurrence came in December of the same year though it was later confirmed that it was a cow of Canadian origin and imported to the U.S. Canada announced two additional cases of BSE from Alberta in early 2005. In June 2005 Dr. John Clifford, chief veterinary officer for the United States Department of Agriculture animal health inspection service confirmed a fully domestic case of BSE in Texas. Dr. Clifford would not identify the ranch, calling that "privileged information." The 12 year old animal was alive at the time when Oprah Winfrey raised concerns about cannibalistic feeding practices on her show which aired April 16, 1996.
https://wn.com/Bse_And_Cjd_In_The_U.K._Explained
Human BSE and compensation for CJD families
10:36

Human BSE and compensation for CJD families

  • Order:
  • Duration: 10:36
  • Updated: 04 Dec 2008
  • views: 514
videos
Click herehttp://www.foodsafetypolicy.com
https://wn.com/Human_Bse_And_Compensation_For_Cjd_Families
Theory on the origins of BSE (2005): United Kingdom video
9:57

Theory on the origins of BSE (2005): United Kingdom video

  • Order:
  • Duration: 9:57
  • Updated: 30 Dec 2008
  • views: 4723
videos
Click herehttp://www.foodsafetypolicy.com New variant CJD may have come from human remains which were put into cattle feed. This theory has been criticised but it is some interesting research into the causes of BSE. *** CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts. The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordination dysfunction (ataxia), changes in gait, rigid posture, and seizures. The duration of the disease varies greatly, but sporadic (non-inherited) CJD can be fatal within months or even weeks. In some people, the symptoms can continue for years. In most patients, these symptoms are followed by involuntary movements and the appearance of an atypical diagnostic electroencephalogram tracing. Most victims die 6 months after initial symptoms appear, often of pneumonia due to impaired coughing reflexes. About 15% of patients survive 2 or more years. The symptoms of CJD are caused by the progressive death of the brain's nerve cells, which is associated with the build-up of abnormal prion proteins forming amyloids. When brain tissue from a CJD patient is examined under a microscope, many tiny holes can be seen where whole areas of nerve cells have died. The word "spongiform" in "transmissible spongiform encephalopathies" refers to the sponge-like appearance of the brain tissue. Cause Transmissible spongiform encephalopathy diseases are caused by prions. The diseases are thus sometimes called prion diseases. Other prion diseases include Gerstmann--Sträussler--Scheinker syndrome (GSS), fatal familial insomnia (FFI) and kuru in humans, as well as bovine spongiform encephalopathy (BSE, commonly known as mad cow disease) in cattle, chronic wasting disease (CWD) in elk and deer, and scrapie in sheep. Alpers' syndrome in infants is also thought to be a transmissible spongiform encephalopathy caused by a prion.
https://wn.com/Theory_On_The_Origins_Of_Bse_(2005)_United_Kingdom_Video
Human 'Mad Cow Disease' (CJD) News Report
3:17

Human 'Mad Cow Disease' (CJD) News Report

  • Order:
  • Duration: 3:17
  • Updated: 29 Nov 2008
  • views: 29820
videos
Click herehttp://www.foodsafetypolicy.com *** On 16th December 2007, at just 24 years of age, Andy Black died after suffering from the human form of mad cow's disease vCJD (variant Creutzfeldt-Jakob Disease). Andy worked in the media producing/researching programmes for talkSPORT Radio, BBC, and ITV. During his terrible last days and at his request Christine Lord made it her mission to find out "Who killed my son?" and with the BBC, she produced a documentary exposing the key players at the centre of the BSE crisis whose actions and decisions led to his untimely and avoidable death. The Documentary is at http://www.youtube.com/watch?v=MAJTr6Nxxa0 Christine Lord, as Andy's mum and a freelance journalist continues to research the scandal - and its links between school meals, (state and independent), baby food, vaccines and medicines. *** Creutzfeldt--Jakob disease or CJD is a degenerative neurological disorder (brain disease) that is incurable and invariably fatal. CJD is at times called a human form of mad cow disease, given that bovine spongiform encephalopathy is believed to be the cause of variant Creutzfeldt--Jakob disease in humans. CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts. The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordination dysfunction (ataxia), changes in gait, rigid posture, and seizures. The duration of the disease varies greatly, but sporadic (non-inherited) CJD can be fatal within months or even weeks (Johnson, 1998). In some people, the symptoms can continue for years. In most patients, these symptoms are followed by involuntary movements and the appearance of an atypical diagnostic electroencephalogram tracing. Most victims die 6 months after initial symptoms appear, often of pneumonia due to impaired coughing reflexes. About 15% of patients survive 2 or more years. The symptoms of CJD are caused by the progressive death of the brain's nerve cells, which is associated with the build-up of abnormal prion proteins forming amyloids. When brain tissue from a CJD patient is examined under a microscope, many tiny holes can be seen where whole areas of nerve cells have died. The word "spongiform" in "transmissible spongiform encephalopathies" refers to the sponge-like appearance of the brain tissue.
https://wn.com/Human_'Mad_Cow_Disease'_(Cjd)_News_Report
Detecting Prions and Diagnosing Prion Diseases by Byron Caughey
33:54

Detecting Prions and Diagnosing Prion Diseases by Byron Caughey

  • Order:
  • Duration: 33:54
  • Updated: 25 Aug 2015
  • views: 3488
videos
"Progress in Detecting Prions and Diagnosing Prion Diseases" is lectured at the CJD foundation 2015 conference by Byron Caughey. Discussing Creutzfeldt-Jakob Disease (CJD) and other Prion diseases, Bryon Caughey helps shed some light on these health issues. Byron Caughey is from the Laboratory of Persistent Viral Diseases, NIH/NIAID Rocky Mountain Laboratories, Hamilton, Montana. Caughey discusses the progress being made to diagnose various protein misfolding diseases in living humans. Topics discussed include: prion diseases, transmissible spongiform encephalopathies, chronic wasting disease, neurodegenerative diseases, bank voles, sporadic CJD,familial CJD, scrapie, GSS, fatal familial insomnia, iatrogenic CJD,. dura mater transplant, growth hormone injections, kuru in New Guinea, protozoans, electron micrographs, brain, PrPC, Christina Orru, RT Quic, RT-quic, assay testing, Real-time quaking induced conversion, amyloid structure, Alzheimers, cerebral spinal fluid or CSF, neurosurgery, neurosurgeries, brushing of nasal mucosa, Montana, Luigi Zanusso, nasal vault, diagnostic sensitivity, specificity, sensor molecules, substrates, substrate, bank vole prion, pathological protein, BSE, Bovine spongiform encephalopathy. 0:00 - Introduction 1:12 - Transmissible Spongiform Encephalopathies Diseases 1:49 - Human TSE (prion) diseases 5:54 - How Prions Propagate 11:11 - Detecting Prion Diseases 17:57 - RT-QuiC tests for TSE Prions 29:58 - Diagnosing Prion Disease Conclusion 30:30 - Future Prospects of Detecting Prion Diseases -- Watch more from this conference at: 2015 CJD Foundation Family Conference Playlist: http://bit.ly/cjd-2015 Individual Presentations: Byron Caughey - Detecting Prions: http://bit.ly/cjd-caughey Glenn Telling - Transgenic Mouse Modeling: http://bit.ly/cjd-telling Thomas Wisniewski - Vaccination Approaches: http://bit.ly/cjd-wisniewski Q&A with Caughey, Telling, Wisniewski, Will: http://bit.ly/cjd-qa1 Jean-Phillipe Deslys: Human Mini-Brains: http://bit.ly/cjd-deslys Julia Moreno: Gerstamann-Straussler Scheinker Syndrome: http://bit.ly/cjd-moreno Chrsitina Sigurdson - Selective Cell Vulnerability: http://bit.ly/cjd-sigurdson Q&A with Deslys, Moreno, Sigurdson, and Knight: http://bit.ly/cjd-qa2 Emiliano Biasini - Pharmacological Chaperones http://bit.ly/cjd-biasini CJD Foundation Website: http://www.CJDFoundation.org Subscribe: https://goo.gl/hRMXAp -- "Prion diseases are a group of rare, invariably fatal brain disorders which occur both in humans and certain animals. They first came to public attention in the mid 1980s in the form of the BSE epidemic in the United Kingdom. BSE (bovine spongiform encephalopathy) is a prion disease in cattle. Tissue from infected animals may have contaminated cattle feed, leading to the silent spread of the BSE epidemic. There is also a theory that BSE came from feed contaminated with scrapie, the long established sheep prion disease. Inevitably, concern over whether BSE could pass to humans mounted. In humans the best known of the prion diseases is Creutzfeldt-Jakob Disease (CJD), which reportedly affects around one person per million per year. In the United States this translates to 250-300 new cases per year. It is well known that CJD is very difficult to diagnose leading to speculation that the one case per million report may be incorrect. Most of the cases are "classical" or "sporadic" CJD (sCJD), occurring for no, as yet, known reason. The sporadic form accounts for approximately 85% of the cases, the familial form approximately 15%. There have also been a few cases which have occurred from contamination via medical procedures; this type is known as iatrogenic or Acquired CJD. Finally over the last few years, another type of Acquired CJD called variant (vCJD) has been identified in young people. CJD has been linked to ingestion of beef tainted with BSE (bovine spongiform encephalopathy), most cases have occurred in the United Kingdom." -- Information Provided by: http://www.cjdfoundation.org/about-cjd
https://wn.com/Detecting_Prions_And_Diagnosing_Prion_Diseases_By_Byron_Caughey
BSE - Clinical Signs - Sensory Disturbances
2:13

BSE - Clinical Signs - Sensory Disturbances

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  • Duration: 2:13
  • Updated: 03 Dec 2014
  • views: 13186
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https://wn.com/Bse_Clinical_Signs_Sensory_Disturbances
Bovine spongiform encephalopathy
38:20

Bovine spongiform encephalopathy

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  • Duration: 38:20
  • Updated: 20 Feb 2013
  • views: 49886
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Bovine spongiform encephalopathy (BSE) is characterized by behavioural changes, locomotory disturbaances and loss of condition. The course of the disease is about 1-3 months terminating in death in some animals, but many are slaughtered because of unmanageable behaviour or trauma due to failling. Affected cattle were first identified in the United Kingdom in 1986, as a result of feeding contaminated ruminant protein and affected nearly 200 000 cattle. Since then BSE has been reported in several other countries. Variant Creutzfeldt-Jacob disease in humans most likely results from the consumption of beef products contaminated by central nervous system tissue. The complete learning module is available through the African Veterinary Information Portal (AfriVIP) at: http://www.afrivip.org/education/livestock/high-impact/contagious-diseases/bovine-spongiform-encephalopathy/2012. AfriVIP is not responsible for verifying the proprietary content of the materials. Any veterinary medical information in this material is intended to inform and educate and is not a tool for diagnosis or a replacement for veterinary evaluation, advice, diagnosis or treatment by a veterinary professional. Viewer discretion is advised: Some veterinary content is graphic and may not be suitable for all viewers. Copyright: University of Pretoria. The copyright of this video was purchased from Prof Dr med. Vet. Ueli Braun, Department für Nutztiere der Universität Zürich, Winterthuresrstrasse 260, CH-8057, Zürich
https://wn.com/Bovine_Spongiform_Encephalopathy
BSE - UK
20:41

BSE - UK

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  • Duration: 20:41
  • Updated: 07 Nov 2007
  • views: 11256
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July 2005 The cost of the crisis over BSE, and its links to the human variant CJD were obvious to all in terms of cattle and livelihood. However, the question of the role that cost had to play before the crisis erupted is one that is much more contentious. Ron McCullagh illustrates both the devastating impact of the crisis, and the debates that still rage over the nature and treatment of the disease.
https://wn.com/Bse_UK
Hyperekplexia as the presenting symptom of Creutzfeldt-Jakob disease
0:32

Hyperekplexia as the presenting symptom of Creutzfeldt-Jakob disease

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  • Duration: 0:32
  • Updated: 09 Dec 2015
  • views: 17538
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We describe a patient with hyperekplexia as the initial presenting symptom in sporadic Creutzfeldt-Jakob disease (CJD). We also demonstrate the utility of the sensitive and highly specific new prion protein amplification assay, real-time quaking-induced conversion (RT-QUIC), which amplifies and detects misfolded β-sheet–rich conformers of the prion protein (PrPsc), in expediting a confident clinical diagnosis of CJD in an atypical case. For more, see: http://cp.neurology.org/content/5/6/498.extract
https://wn.com/Hyperekplexia_As_The_Presenting_Symptom_Of_Creutzfeldt_Jakob_Disease
Creutzfeldt-Jakob disease (CJD)
2:01

Creutzfeldt-Jakob disease (CJD)

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  • Duration: 2:01
  • Updated: 21 Aug 2008
  • views: 411880
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Video of a patient suffering from Creutzfeldt-Jakob disease (CJD). For Doctors - Visit http://doctorshangout.com/video/video/show?id=2002836%3AVideo%3A92991 For Patients - Join http://www.patient.org.in - It is a health based social network of people sharing their disease experiences, treatment options, knowledge and giving support to other people suffering from similar health problems.
https://wn.com/Creutzfeldt_Jakob_Disease_(Cjd)
Part 1: Mad Cow Disease and Food Safety News Programme
5:37

Part 1: Mad Cow Disease and Food Safety News Programme

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  • Duration: 5:37
  • Updated: 15 Dec 2008
  • views: 33117
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Click herehttp://www.foodsafetypolicy.com/documentary BBC Documentary about Andy Black (from Portsmouth, England) who died after suffering from the human form of mad cow disease variant CJD. vCJd fact file Creutzfeldt-Jakob disease is a rare and fatal progressive degenerative brain disease. It is one of a group of diseases which affect humans and animals called Transmissible Spongiform Encephalopathies (TSEs). TSEs are believed to be caused by the build up of an abnormal form of the naturally occurring 'prion' protein in the brain. CJD was originally described in its classical form in 1920. A new variant - called variant CJD (vCJD) - was first identified in 1996. Variant CJD is strongly linked to exposure to a TSE of cattle called Bovine Spongiform Encephalopathy (BSE), probably through food. Moving and harrowing story We started filming Christine Lord in September 2007, just a couple of months after her son Andrew was diagnosed with variant Creutzfeldt Jakob Disease (vCJd). Andrew had been a fit and healthy radio producer, but the disease was already damaging his mind and body. He was unable to walk more than a few steps and struggled to speak above a whisper. Christine borrowed one of our cameras, so that she could film Andrew at home and record her thoughts. These video diaries provided a moving insight into the harrowing months that followed as Christine cared for her son. Christine's a freelance journalist, so she used her spare moments to look into the history of BSE and the possible causes of vCJD. While Andrew slept, she researched the disease that was killing him. Voice in the wilderness? We also followed Christine as she visited Pitsham Farm near Midhurst, where the first cow was spotted with Bovine Spongiform Encephalopathy (BSE) in 1984. The farm is just 30 miles away from Christine's home in Southsea, but it was the furthest she had been from Andrew since he was diagnosed. Emotional meeting - Christine and David. Vet David Bee told Christine how he labelled the disease "Pitsham Farm Syndrome" after seeing eight cows with similar symptoms. "We had a range of clinical symptoms. We had cows that were aggressive, cows that had lost weight and cows with head tremors. And we weren't seeing those anywhere else." For Christine, the vet's description is distressingly familiar: "The symptoms you have described are exactly what I have been experiencing with my son. "I have to be his voice in the wilderness because it feels like we've been forgotten, that the broader public have forgotten about it, but everyone over the age of 10 has been exposed to this." Search for the truth Christine's search for the truth also took her to London to see the man leading the search for the cure. Search for truth - Professor Collinge. Professor John Collinge and his team at the Prion Unit have managed to reverse the disease in mice. It's too late for Andrew, but Professor Collinge thinks a similar treatment for humans might be just a few years away. He also tells Christine how the government "leaned on" scientists who tried to speak out about the health risks of BSE in the early '90s. "I had phone calls from government departments in somewhat intimidating tones. They left you in no doubt that you weren't making yourself very popular." Confronting the politicians Christine's most testing encounter was with the politician who will always be associated with the BSE crisis. Agriculture Minister John Gummer attempted to convince the public that beef was safe by feeding a burger to his daughter Cordelia. Safe to eat? John Gummer with Cordelia. The BSE inquiry concluded that the government's campaign of reassurance was a mistake, but it didn't criticise Mr Gummer for his role in the crisis. At a highly charged meeting, Mr Gummer tells Christine that he took the right decisions based on the knowledge he had at the time. "At every point and on every occasion, I sought to make the best choice and best decision I could for the safety of myself, my family and my country." He strongly denies Christine's suggestion that the Government relied on selective scientific research. "No scientist of any kind was stopped by me from giving their information. I only wanted to know the facts."
https://wn.com/Part_1_Mad_Cow_Disease_And_Food_Safety_News_Programme
Ministerial Statements on BSE and CJD: 1990 - 2000
1:42

Ministerial Statements on BSE and CJD: 1990 - 2000

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  • Duration: 1:42
  • Updated: 16 Aug 2010
  • views: 479
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Click herehttp://www.foodsafetypolicy.com Ministerial Statements on U.K. BSE and CJD: 1990 - 2000 1) Agriculture Minister from 1990 2) Prime Minister from 1995 3) Health Minister from 1996 4) Agriculture Minister from 2000
https://wn.com/Ministerial_Statements_On_Bse_And_Cjd_1990_2000
What causes BSE, CJD & MS? Organic Farmer Mark Purdey on Organophosphates (2001)
36:37

What causes BSE, CJD & MS? Organic Farmer Mark Purdey on Organophosphates (2001)

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  • Duration: 36:37
  • Updated: 16 Mar 2011
  • views: 6814
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Mark Purdey and Organophosphate (Film/Video, i-Contact, September 2001) Organic livestock farmer Mark Purdey was ordered in the 1980's, along with all cattle and dairy farmers in the UK, to treat his cows with an organophosphate pesticide 'Phosmet' manufactured by I.C.I.. Organophosphates are derived from military nerve gas and a systemic treatment (the chemical enters the entire internal system of the cow) would undermine his organic principles so Mark refused to treat. "If Phosmet is proven to have caused BSE, the worldwide use of organophosphates (OPs) could be put into jeopardy, costing the chemical industry billions. The government know more than they're letting on. They've stuck to the scrapie theory to placate people and give the impression they've got it under control." Mark Purdey, Organic Dairy Farmer, Exmoor. "If the government are found liable for BSE - by enforcing organophosphate treatment - the payout could break the economy." Tom King (Purdey's MP) Leigh Day & Co are the legal firm Mark mentions with reference to the 1988 Camelford Water Disaster 30 minites in: http://www.911forum.org.uk/board/viewtopic.php?t=21090 Whoever the monkeys have been at the top of the tree, the Party line has stayed the same. Mad Cow Disease came about by feeding scrapie infected meat and bone meal to cows. But one West Country farmer has a different theory. One that the authorities and the pesticide producers have gone to great to lengths to silence. Between the late 70's and 1982 British farmers were forced by law to treat their cows for warble fly with a pour on organophosphate called phosmet - organophosphates are derived from nerve gas formulated by nazi chemists during World War II. Big business soon realised its profit potential and, post war, it was exclusively marketed as an agricultural pesticide by ICI, and later their cunningly renamed subdivision Zeneca. Seeing how his own organically reared cows never developed BSE, but phosmet-treated cattle brought onto the farm did, Somerset dairy farmer Mark Purdey refused to treat his herd. In 1984 MAFF took him to the High Court, but lost. "Before 1982 farmers could treat warbles with an organic ground-up root compound called Derris. This was outlawed, so they could sell more organophosphates," said Purdey. Organophosphates, used to treat headlice in school children, have been implicated as a potential cause of Gulf War Syndrome. Purdey managed to alleviate symptoms in a BSE infected cow by injecting oxime, an antidote to pesticide poisoning. The cure was never completed as MAFF turned up and destroyed the cow. Unconvinced by the accepted cause of BSE and CJD, Purdey set about studying how disease clusters reflected OP usage. He found Britain, the only country enforcing phosmet use, to have the highest rate of disease. Ireland had some BSE, but OP use was voluntary, and given at a lower dose. Brittany (France) began to develop BSE following an enforced phosmet trial, and human new variant CJD was clustered in Kent's Wield Valley, where hop and top fruit growth gets saturated with organophosphates. Agitated by Purdey's discoveries, the pesticide industry hit back. The dubiously named National Office of Animal Health (NOAH), a lobby group representing the UK animal medicine industry, whose membership reads like a Downing St dinner party invite list of extremely dodgy chemical interests - including Bayer, Monsanto, Novartis, Pfizer, Roche, Schering-Plough etc - published documents discrediting Purdey's work. NOAH produced an independent expert, Dr David Ray, for the BSE Inquiry, who turned out to be receiving funding from Zeneca for his Medical Research Council toxicology unit. "I don't think this affected my judgement," Ray told SchNEWS. "You may not believe it, but I didn't realise Zeneca produced phosmet at the time." Hmmm. In March 1996 - one week before the UK government admitted to a link between BSE and new variant CJD - Zeneca sold the phosmet patent to a PO Box company in the Arizona desert. As Ray said: "Zeneca are not keen to be sued." http://www.cultureshop.org/details.php?code=PURDEY For a more in depth view of this story and the full dirt on David Ray and the 'intellectual corruption' that's rife in the research and licensing of veterinary medicines see www.squall.co.uk
https://wn.com/What_Causes_Bse,_Cjd_Ms_Organic_Farmer_Mark_Purdey_On_Organophosphates_(2001)
Creutzfeldt-Jakob Disease Report from 2010
1:35

Creutzfeldt-Jakob Disease Report from 2010

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  • Duration: 1:35
  • Updated: 12 Sep 2011
  • views: 3227
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See http://www.foodsafetypolicy.com CJD Research Campaign from October 2011 Two decades on since BSE or mad cow disease hit the headlines, a father of a victim of its form in humans, new variant CJD, wants the government to maintain research into the degenerative condition. Recent research suggests up to 1 in 1000 people could still be affected. *** Creutzfeldt--Jakob disease or CJD is a degenerative neurological disorder (brain disease) that is incurable and invariably fatal.CJD is at times called a human form of mad cow disease, given that bovine spongiform encephalopathy is believed to be the cause of variant Creutzfeldt--Jakob disease in humans. CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordination dysfunction (ataxia), changes in gait, rigid posture, and seizures. The duration of the disease varies greatly, but sporadic (non-inherited) CJD can be fatal within months or even weeks (Johnson, 1998). In some people, the symptoms can continue for years. In most patients, these symptoms are followed by involuntary movements and the appearance of an atypical diagnostic electroencephalogram tracing. Most victims die 6 months after initial symptoms appear, often of pneumonia due to impaired coughing reflexes. About 15% of patients survive 2 or more years. The symptoms of CJD are caused by the progressive death of the brain's nerve cells, which is associated with the build-up of abnormal prion proteins forming amyloids. When brain tissue from a CJD patient is examined under a microscope, many tiny holes can be seen where whole areas of nerve cells have died. The word "spongiform" in "transmissible spongiform encephalopathies" refers to the sponge-like appearance of the brain tissue. Cause Transmissible spongiform encephalopathy diseases are caused by prions. The diseases are thus sometimes called prion diseases. Other prion diseases include Gerstmann--Sträussler--Scheinker syndrome (GSS), fatal familial insomnia (FFI) and kuru in humans, as well as bovine spongiform encephalopathy (BSE, commonly known as mad cow disease) in cattle, chronic wasting disease (CWD) in elk and deer, and scrapie in sheep. Alpers' syndrome in infants is also thought to be a transmissible spongiform encephalopathy caused by a prion.
https://wn.com/Creutzfeldt_Jakob_Disease_Report_From_2010
Mad Cow Disease and Food Safety News Programme (Full length)
29:13

Mad Cow Disease and Food Safety News Programme (Full length)

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  • Duration: 29:13
  • Updated: 01 Jul 2011
  • views: 18750
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See http://www.foodsafetypolicy.com/documentary for the interview towards the end of the programme. Joe Crowley's (the presenter's) filming diary... For me and friends of my age, Mad Cow Disease seems like a distant nightmare - something that haunted us on the news as we grew up. But this Inside Out programme has made me think again and, sadly, given the disease a painful new relevance. It's about a young guy like me, starting out on a career in the media, when he was struck down by the human form of the disease. And it's a story of his mother's (Christine Lord) relentless search for answers, as she tries to find out who killed her son (Andrew Black). *** Further informarion on variant CJD Inside Out looks at the rise of Variant CJD, the symptoms and the support available for those caring for or mourning the loss of loved ones from the disease. Variant CJD Variant CJD is a form of transmissible dementia and differs from other forms of CJD. It has received a lot of media attention over recent years, but has been around for much longer. Early 1920s CJD is first recognised by two German neurologists Creuzfeldt and Jakob, the namesakes of the disease. Following decades It is a rare disease with an estimated UK occurrence of 30-50 cases per year. Dec 1984 Cow number 133 on Stent farm Sussex becomes the first confirmed victim of BSE the animal strain of CJD. Feb 1985 Cow 133 dies. Nov 1986 BSE is first formally identified by the Central Veterinary Laboratory. April 1988 The Government establishes the Southwood committee to look into BSE. It concludes that BSE is probably spread in animal feed. May 1990 Chief Medical Officer says beef is safe to eat. July 1993 100,000th case of BSE in Britain. May 1995 First death from Variant CJD. March 1996 Government announces suspected link between BSE and the human equivalent CJD . The EU announces worldwide export ban on all British beef. March 1998 A public inquiry into the origin and spread of BSE and CJD opens in London. Jan 2000 Claire Mcvey, age 15, dies of Variant CJD. August 2002 92 deaths in Britain to date from definite Variant CJD . 22 deaths from probable Variant CJD. Symptoms Although symptoms are diverse across patients, a number of similarities have been found when sufferers' brain were examined under a microscope. According to the Crentzfelt-Jakob Support Network, most had lost nerve cells in the brain leading to a spongy appearance. There are many symptoms of Variant CJD, which can broadly be split into two categories... Physical symptoms: Unsteadiness on feet Development of uncontrollable jerky movements Progressive stiffness of the limbs Incontinence Mental symptoms: The progressive loss of mental abilities Mood swings Loss of awareness of surroundings The incubation period for the disease is years rather than weeks or months. Therefore, the symptoms get progressively worse over time. Medicine According to the Crentzfelt-Jakob Support Network, most people with CJD die within six months. But in about 10 per cent of cases it can last between two and five years. At present, there is no known cure for CJD despite considerable, ongoing research. There are some medicines which make the last few weeks easier for sufferers. Source of information: http://www.bbc.co.uk/insideout/southwest/series1/claire-mcvey.shtml
https://wn.com/Mad_Cow_Disease_And_Food_Safety_News_Programme_(Full_Length)
What is Mad Cow Disease?
1:25

What is Mad Cow Disease?

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  • Duration: 1:25
  • Updated: 30 Apr 2012
  • views: 195104
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Mad Cow Disease - What to Know - as part of the education series by GeoBeats. Mad Cow Disease - What is It? Scientifically named Bovine Spongiform Encephalopathy, it is a progressive neurological disease that affects a cow's nervous system. Once infected, the cow's behavior becomes erratic - inability to walk, uncontrolled movements and lower milk production. Incubation period can be months, even years. There's no known treatment and the disease is fatal. How Do Cows Get Infected? A cow gets infected with the disease if they eat diseased feed which has abnormal proteins called prions found in brains, spinal cords, and other parts. These prions cause the degeneration in brain, giving it a spongy like appearance. How Can It Affect Us? Humans don't get mad cow disease as such but they get a variant of it if they eat the diseased cattle tissue. Health officials in the US and elsewhere control such scenarios by periodic sampling and prohibiting cattle parts in feed which may contain prions. Where Has It Been Reported So Far? Although United Kingdom has been most impacted by the mad cow disease so far, the BSE has been reported in many countries around the word. Millions of cattle have been slaughtered and nearly 200,000 cows have died. Around 200 humans have lost their lives. In the US recently, a cow tested positive for the disease but the health officials say that the infected cow wasn't designated for meat consumption and it was an atypical strain of the disease.
https://wn.com/What_Is_Mad_Cow_Disease
UK: TEENAGER WHO DIED FROM HUMAN EQUIVALENT OF MAD COW DISEASE
1:45

UK: TEENAGER WHO DIED FROM HUMAN EQUIVALENT OF MAD COW DISEASE

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  • Duration: 1:45
  • Updated: 21 Jul 2015
  • views: 893
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English/Nat The parents of a teenager who died from the human equivalent of Mad Cow Disease hit out Thursday at the British government's handling of the issue. Stephen Churchill was 19 when he died last year from Disease. The government has only now admitted there's a link between the two conditions. Their grief dates from a year ago - but their anger is new. Dorothy and David Churchill say the death of their son Stephen had been a mystery - until now. They recall how his illness started. SOUNDBITE: (English) "All the things that you and I do naturally, take for granted, Stephen couldn't do. He couldn't feed himself, he couldn't walk, he had no coordination, his short term memory went, couldn't write and he actually was only in bed the last few days of his life. Prior to that he did get up and sit in a chair but he couldn't walk, he had to be moved in a wheelchair." SUPER CAPTION: Dorothy Churchill, Mother Stephen's father tells how initially the doctors were taken completely by surprise. SOUNDBITE: (English) "Well we were, perhaps even like the medics, we were very confused because when Stephen was in the psychiatric ward they said, well this is a severe depression but with bizarre symptoms. Then when he went to the general hospital it was diagnosed that it was a progressive, degenerative, neurotic disease but they didn't have a name for it. We now know the reasons why, that's because Stephen's age was confusing people. CJD didn't strike people that old it was always the disease of the 50, 60 or 70 year old." SUPER CAPTION: David Churchill, Father Stephen's parents are angry that only now has the UK government accepted there's a link between Mad Cow Disease and the illness he died from - Creutzfeldt- Jakob Disease - and that British beef could be to blame. SOUNDBITE: (English) "Shocked, angry, confused, you know if you'd asked me yesterday I'd say I really don't think there is anything in beef but now we're told Stephen is one of the 10 that was infected through BSE beef and it just makes you so angry." SUPER CAPTION: Dorothy, Stephen's mother Now they want an independent inquiry into the affair. You can license this story through AP Archive: http://www.aparchive.com/metadata/youtube/544050ffccdd8ab62fc60a0e8a5af3d6 Find out more about AP Archive: http://www.aparchive.com/HowWeWork
https://wn.com/UK_Teenager_Who_Died_From_Human_Equivalent_Of_Mad_Cow_Disease
Creutzfeldt-Jakob Disease (CJD) "Mad Cow Disease"- My Father's Story (with captions english-espanol)
5:28

Creutzfeldt-Jakob Disease (CJD) "Mad Cow Disease"- My Father's Story (with captions english-espanol)

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  • Duration: 5:28
  • Updated: 13 Nov 2013
  • views: 11563
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Rafael Alvaro Bohorquez Villamil passed away after a brave 9 weeks from a very rare disease: Creutzfeldt-Jakob Disease (CJD) on October 28, 2013, after only a few months of arriving to Canada as a tourist where he was enjoying his life and good health with his youngest grandson. Everyone he met in Toronto in this short period saw what a great man he was, warm and joyful, full of peace. Everyone that knew him in other parts of the world already knew this about him.Loving father,grandfather, brother, uncle and boss and friend, he will be greatly missed and will remain forever in out hearts. Any donations to help pay hospital bills will be appreciated and cheques can be written directly to the hospital: North York General Hospital BOHORQUEZ VILLAMIL MRN1735438 Or if you would like to use GO FUND ME: http://www.gofundme.com/alvaro A bank account has also been set up for this cause at Scotiabank 92072-002-04722786 John 14:1-4 "Let not your hearts be troubled. Believe in God; believe also in me. In my Father's house are many rooms. If it were not so, would I have told you that I go to prepare a place for you? And if I go and prepare a place for you, I will come again and will take you to myself, that where I am you may be also. And you know the way to where I am going."
https://wn.com/Creutzfeldt_Jakob_Disease_(Cjd)_Mad_Cow_Disease_My_Father's_Story_(With_Captions_English_Espanol)
Mad Cow Disease-vCJD
3:20

Mad Cow Disease-vCJD

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  • Duration: 3:20
  • Updated: 11 Apr 2016
  • views: 926
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https://wn.com/Mad_Cow_Disease_Vcjd