• Creutzfeldt-Jakob disease, Human Mad Cow Disease, Kuru, Scrapies, CJD, BSE

    published: 16 Dec 2016
  • What causes BSE, CJD & MS? Organic Farmer Mark Purdey on Organophosphates (2001)

    Mark Purdey and Organophosphate (Film/Video, i-Contact, September 2001) Organic livestock farmer Mark Purdey was ordered in the 1980's, along with all cattle and dairy farmers in the UK, to treat his cows with an organophosphate pesticide 'Phosmet' manufactured by I.C.I.. Organophosphates are derived from military nerve gas and a systemic treatment (the chemical enters the entire internal system of the cow) would undermine his organic principles so Mark refused to treat. "If Phosmet is proven to have caused BSE, the worldwide use of organophosphates (OPs) could be put into jeopardy, costing the chemical industry billions. The government know more than they're letting on. They've stuck to the scrapie theory to placate people and give the impression they've got it under control." Mark P...

    published: 16 Mar 2011
  • Creutzfeldt-Jakob disease (CJD)

    Video of a patient suffering from Creutzfeldt-Jakob disease (CJD). For Doctors - Visit http://doctorshangout.com/video/video/show?id=2002836%3AVideo%3A92991 For Patients - Join http://www.patient.org.in - It is a health based social network of people sharing their disease experiences, treatment options, knowledge and giving support to other people suffering from similar health problems.

    published: 21 Aug 2008
  • Ministerial Statements on BSE and CJD: 1990 - 2000

    Click herehttp://www.foodsafetypolicy.com Ministerial Statements on U.K. BSE and CJD: 1990 - 2000 1) Agriculture Minister from 1990 2) Prime Minister from 1995 3) Health Minister from 1996 4) Agriculture Minister from 2000

    published: 16 Aug 2010
  • Prion Disease and CJD Vaccination Approaches by Thomas Wisniewski

    Discussing vaccination approaches for Prion Diseases, CJD, and related disorders, Dr. Thomas Wisniewski sheds light on how to solve the dilemmas of Creutzfeldt-Jakob Disease and similar disorders. Thomas Wisniewski: Professor of neurology, Personal Chair in Clinical Neurology, University of Edinburgh; consultant neurologist, Western General Hospital, Founder and Former director, National CJD Surveillance Unit, United Kingdom, Edinburgh, Scotland. 0:00 - Introduction 0:31 - Conformational Disorders 0:59 - Prion Disease Active Vaccination 2:05 - Salmonella Vaccines for Prion Infection 2:54 - Chronic Wasting Disease (CWD) 5:09 - Saliva Sampling and Tonsillar Biopsy 7:37 - Conclusions of Prion Disease Vaccination 8:01 - Immunotherapeutic Approches for AD 16:12 - Conclusions of Monoclonal Ab...

    published: 03 Sep 2015
  • Mad Cow Disease and Food Safety News Programme (Full length)

    See http://www.foodsafetypolicy.com/documentary for the interview towards the end of the programme. Joe Crowley's (the presenter's) filming diary... For me and friends of my age, Mad Cow Disease seems like a distant nightmare - something that haunted us on the news as we grew up. But this Inside Out programme has made me think again and, sadly, given the disease a painful new relevance. It's about a young guy like me, starting out on a career in the media, when he was struck down by the human form of the disease. And it's a story of his mother's (Christine Lord) relentless search for answers, as she tries to find out who killed her son (Andrew Black). *** Further informarion on variant CJD Inside Out looks at the rise of Variant CJD, the symptoms and the support available for thos...

    published: 01 Jul 2011
  • UK: LONDON: BSE CRISIS LATEST

    English/Nat XFA European farm ministers are meeting in Brussels on Monday for crisis talks expected to result in tough new restrictions on cattle feed to control the spread of mad cow disease throughout the continent. The restrictions, set to be imposed throughout the European Union, are more stringent than those introduced by Britain in 1996 following the establishment of a probably link between the disease, also known as BSE, and the lethal human condition variant CJD. The existing ban on meat and bonemeal (MBM) feed for cattle could be extended to all other animals including pigs, chickens and pets, and feed containing fish meal and chicken remains could also be banned. Monday's meeting comes after panic in France, where BSE cases are rising and four human victims of vCJD hav...

    published: 21 Jul 2015
  • BSE symptom video

    published: 02 May 2015
  • BSE - UK

    July 2005 The cost of the crisis over BSE, and its links to the human variant CJD were obvious to all in terms of cattle and livelihood. However, the question of the role that cost had to play before the crisis erupted is one that is much more contentious. Ron McCullagh illustrates both the devastating impact of the crisis, and the debates that still rage over the nature and treatment of the disease.

    published: 07 Nov 2007
  • How, and when, people could get variant CJD?

    Click here http://www.foodsafetypolicy.com/howandwhen Bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, is a fatal neurodegenerative disease in cattle that causes a spongy degeneration in the brain and spinal cord. BSE has a long incubation period, about 30 months to 8 years, usually affecting adult cattle at a peak age onset of four to five years, all breeds being equally susceptible. In the United Kingdom, the country worst affected, more than 180,000 cattle have been infected and 4.4 million slaughtered during the eradication program. The disease may be most easily transmitted to human beings by eating food contaminated with the brain, spinal cord or digestive tract of infected carcasses. However, it should also be noted that the infectious agent, although mo...

    published: 03 Aug 2010
  • Theory on the origins of BSE (2005): United Kingdom video

    Click herehttp://www.foodsafetypolicy.com New variant CJD may have come from human remains which were put into cattle feed. This theory has been criticised but it is some interesting research into the causes of BSE. *** CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts. The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordinati...

    published: 30 Dec 2008
  • UK: TEENAGER WHO DIED FROM HUMAN EQUIVALENT OF MAD COW DISEASE

    English/Nat The parents of a teenager who died from the human equivalent of Mad Cow Disease hit out Thursday at the British government's handling of the issue. Stephen Churchill was 19 when he died last year from Disease. The government has only now admitted there's a link between the two conditions. Their grief dates from a year ago - but their anger is new. Dorothy and David Churchill say the death of their son Stephen had been a mystery - until now. They recall how his illness started. SOUNDBITE: (English) "All the things that you and I do naturally, take for granted, Stephen couldn't do. He couldn't feed himself, he couldn't walk, he had no coordination, his short term memory went, couldn't write and he actually was only in bed the last few days of his life...

    published: 21 Jul 2015
  • Prion Disease | Gerstmann-Straussler Scheinker (GSS) Syndrome by Julia Moreno

    Discussing the specific Prion Disease, Gerstmann-Straussler Scheinker (GSS) Syndrome, Julia Moreno showcases her studies on the role of Cellular Chaperons, and the Unfolded Protein Response in the Gerstmann-Straussler Scheinker (GSS) Syndrome. 0:30 - Human Prion Disease Types 1:29 - Gerstmann-Straussier Scheinker (GSS) 2:30 - Spontaneous Neurodegenerative Disease in Tg(GSS)mice 4:15 - Mis-folds During Prion Disease 5:57 - Hypothesis 6:15 - Generation of Susceptible (S) and Resistant (R) Cells Study 7:46 - Identification of Differences between S and R Cells 9:07 - PANTHER Analysis 15:27 - Conclusions -- Watch more from this conference at: 2015 CJD Foundation Family Conference Playlist: http://bit.ly/cjd-2015 Individual Presentations: Byron Caughey - Detecting Prions: http://bit.ly/cjd-cau...

    published: 03 Sep 2015
  • Human 'Mad Cow Disease' (CJD) News Report No. 2

    Click herehttp://www.foodsafetypolicy.com On December 2007, at just 24 years of age, Andy Black died after suffering from the human form of mad cow's disease vCJD (variant Creutzfeldt-Jakob Disease). Andy worked in the media producing/researching programmes for talkSPORT Radio, BBC, and ITV. During his terrible last days and at his request Christine Lord made it her mission to find out "Who killed my son?" and with the BBC, she produced a documentary exposing the key players at the centre of the BSE crisis whose actions and decisions led to his untimely and avoidable death. The Documentary is at http://www.youtube.com/watch?v=MAJTr6Nxxa0 Christine Lord, as Andy's mum and a freelance journalist continues to research the scandal - and its links between school meals, (state and indepen...

    published: 01 Oct 2013
  • Creutzfeldt-Jakob Disease (CJD) Q&A | Byron Caughey, Glenn Telling, Thomas Wisniewski, Robert Will

    During the Creutzfeldt-Jakob Disease (CJD) question and answer discussion board, leading experts on Prion Disease and similar disorders comment on what CJD is, discuss vaccination approaches to Prion Disease and talk about other aspects of Creutzfeldt-Jakob disease. Prion Disease and CJD Discussion Board: Byron Caughey, Glenn Telling, Thomas Wisniewski, and Robert Will 0:34 - Testing CJD Disease Results 1:31 - Creutzfeldt-Jakob Disease Test Turnout Time 5:00 - CJD Infection Analysis 8:42 - Pre-CJD Symptoms 14:11 - CWD Prevention 22:35 - CWD Immunity 27:24 - How to Detect CWD and Related Diseases 31:01 - Early Detection of CJD and Prion Disease 36:37 - Learning About CJD Through Similar Diseases -- Watch more from this conference at: 2015 CJD Foundation Family Conference Playlist: http:/...

    published: 03 Sep 2015
  • Mad Cow Disease

    Also known as Creutzfeldt Jakob Disease in humans, it will attack your brain and body with deadly, incurable symptoms. Subscribe for new videos: http://goo.gl/SaufF4 Watch the scorpion video here: http://goo.gl/MYiSLm Watch the Brazilian wandering spider video here: http://goo.gl/Z6dFJS Infection from this disease always leads to death…and there is no cure. Today's video was produced due to high demand by our subscribers. If you have any other topics you'd like to learn about, be sure to write your suggestions below What is it? Mad cow disease, or bovine spongiform encephalopathy (BSE), is a disease that affects a cow's nervous system by putting holes in its brain, causing it to become spongy and start wasting away. The disease can also affect other tissues throughout the cow's body as...

    published: 06 Mar 2015
Creutzfeldt-Jakob disease, Human Mad Cow Disease, Kuru, Scrapies, CJD, BSE

Creutzfeldt-Jakob disease, Human Mad Cow Disease, Kuru, Scrapies, CJD, BSE

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  • Duration: 52:11
  • Updated: 16 Dec 2016
  • views: 2989
videos
https://wn.com/Creutzfeldt_Jakob_Disease,_Human_Mad_Cow_Disease,_Kuru,_Scrapies,_Cjd,_Bse
What causes BSE, CJD & MS? Organic Farmer Mark Purdey on Organophosphates (2001)

What causes BSE, CJD & MS? Organic Farmer Mark Purdey on Organophosphates (2001)

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  • Duration: 36:37
  • Updated: 16 Mar 2011
  • views: 6270
videos
Mark Purdey and Organophosphate (Film/Video, i-Contact, September 2001) Organic livestock farmer Mark Purdey was ordered in the 1980's, along with all cattle and dairy farmers in the UK, to treat his cows with an organophosphate pesticide 'Phosmet' manufactured by I.C.I.. Organophosphates are derived from military nerve gas and a systemic treatment (the chemical enters the entire internal system of the cow) would undermine his organic principles so Mark refused to treat. "If Phosmet is proven to have caused BSE, the worldwide use of organophosphates (OPs) could be put into jeopardy, costing the chemical industry billions. The government know more than they're letting on. They've stuck to the scrapie theory to placate people and give the impression they've got it under control." Mark Purdey, Organic Dairy Farmer, Exmoor. "If the government are found liable for BSE - by enforcing organophosphate treatment - the payout could break the economy." Tom King (Purdey's MP) Leigh Day & Co are the legal firm Mark mentions with reference to the 1988 Camelford Water Disaster 30 minites in: http://www.911forum.org.uk/board/viewtopic.php?t=21090 Whoever the monkeys have been at the top of the tree, the Party line has stayed the same. Mad Cow Disease came about by feeding scrapie infected meat and bone meal to cows. But one West Country farmer has a different theory. One that the authorities and the pesticide producers have gone to great to lengths to silence. Between the late 70's and 1982 British farmers were forced by law to treat their cows for warble fly with a pour on organophosphate called phosmet - organophosphates are derived from nerve gas formulated by nazi chemists during World War II. Big business soon realised its profit potential and, post war, it was exclusively marketed as an agricultural pesticide by ICI, and later their cunningly renamed subdivision Zeneca. Seeing how his own organically reared cows never developed BSE, but phosmet-treated cattle brought onto the farm did, Somerset dairy farmer Mark Purdey refused to treat his herd. In 1984 MAFF took him to the High Court, but lost. "Before 1982 farmers could treat warbles with an organic ground-up root compound called Derris. This was outlawed, so they could sell more organophosphates," said Purdey. Organophosphates, used to treat headlice in school children, have been implicated as a potential cause of Gulf War Syndrome. Purdey managed to alleviate symptoms in a BSE infected cow by injecting oxime, an antidote to pesticide poisoning. The cure was never completed as MAFF turned up and destroyed the cow. Unconvinced by the accepted cause of BSE and CJD, Purdey set about studying how disease clusters reflected OP usage. He found Britain, the only country enforcing phosmet use, to have the highest rate of disease. Ireland had some BSE, but OP use was voluntary, and given at a lower dose. Brittany (France) began to develop BSE following an enforced phosmet trial, and human new variant CJD was clustered in Kent's Wield Valley, where hop and top fruit growth gets saturated with organophosphates. Agitated by Purdey's discoveries, the pesticide industry hit back. The dubiously named National Office of Animal Health (NOAH), a lobby group representing the UK animal medicine industry, whose membership reads like a Downing St dinner party invite list of extremely dodgy chemical interests - including Bayer, Monsanto, Novartis, Pfizer, Roche, Schering-Plough etc - published documents discrediting Purdey's work. NOAH produced an independent expert, Dr David Ray, for the BSE Inquiry, who turned out to be receiving funding from Zeneca for his Medical Research Council toxicology unit. "I don't think this affected my judgement," Ray told SchNEWS. "You may not believe it, but I didn't realise Zeneca produced phosmet at the time." Hmmm. In March 1996 - one week before the UK government admitted to a link between BSE and new variant CJD - Zeneca sold the phosmet patent to a PO Box company in the Arizona desert. As Ray said: "Zeneca are not keen to be sued." http://www.cultureshop.org/details.php?code=PURDEY For a more in depth view of this story and the full dirt on David Ray and the 'intellectual corruption' that's rife in the research and licensing of veterinary medicines see www.squall.co.uk
https://wn.com/What_Causes_Bse,_Cjd_Ms_Organic_Farmer_Mark_Purdey_On_Organophosphates_(2001)
Creutzfeldt-Jakob disease (CJD)

Creutzfeldt-Jakob disease (CJD)

  • Order:
  • Duration: 2:01
  • Updated: 21 Aug 2008
  • views: 370150
videos
Video of a patient suffering from Creutzfeldt-Jakob disease (CJD). For Doctors - Visit http://doctorshangout.com/video/video/show?id=2002836%3AVideo%3A92991 For Patients - Join http://www.patient.org.in - It is a health based social network of people sharing their disease experiences, treatment options, knowledge and giving support to other people suffering from similar health problems.
https://wn.com/Creutzfeldt_Jakob_Disease_(Cjd)
Ministerial Statements on BSE and CJD: 1990 - 2000

Ministerial Statements on BSE and CJD: 1990 - 2000

  • Order:
  • Duration: 1:42
  • Updated: 16 Aug 2010
  • views: 454
videos
Click herehttp://www.foodsafetypolicy.com Ministerial Statements on U.K. BSE and CJD: 1990 - 2000 1) Agriculture Minister from 1990 2) Prime Minister from 1995 3) Health Minister from 1996 4) Agriculture Minister from 2000
https://wn.com/Ministerial_Statements_On_Bse_And_Cjd_1990_2000
Prion Disease and CJD Vaccination Approaches by Thomas Wisniewski

Prion Disease and CJD Vaccination Approaches by Thomas Wisniewski

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  • Duration: 17:48
  • Updated: 03 Sep 2015
  • views: 545
videos
Discussing vaccination approaches for Prion Diseases, CJD, and related disorders, Dr. Thomas Wisniewski sheds light on how to solve the dilemmas of Creutzfeldt-Jakob Disease and similar disorders. Thomas Wisniewski: Professor of neurology, Personal Chair in Clinical Neurology, University of Edinburgh; consultant neurologist, Western General Hospital, Founder and Former director, National CJD Surveillance Unit, United Kingdom, Edinburgh, Scotland. 0:00 - Introduction 0:31 - Conformational Disorders 0:59 - Prion Disease Active Vaccination 2:05 - Salmonella Vaccines for Prion Infection 2:54 - Chronic Wasting Disease (CWD) 5:09 - Saliva Sampling and Tonsillar Biopsy 7:37 - Conclusions of Prion Disease Vaccination 8:01 - Immunotherapeutic Approches for AD 16:12 - Conclusions of Monoclonal Ab Experiments 16:38 - Conformational Disorders (Alzheimer's Disease) Therapy Watch more from this conference at: 2015 CJD Foundation Family Conference Playlist: http://bit.ly/cjd-2015 Individual Presentations: Byron Caughey - Detecting Prions: http://bit.ly/cjd-caughey Glenn Telling - Transgenic Mouse Modeling: http://bit.ly/cjd-telling Thomas Wisniewski - Vaccination Approaches: http://bit.ly/cjd-wisniewski Q&A with Caughey, Telling, Wisniewski, Will: http://bit.ly/cjd-qa1 Jean-Phillipe Deslys: Human Mini-Brains: http://bit.ly/cjd-deslys Julia Moreno: Gerstamann-Straussler Scheinker Syndrome: http://bit.ly/cjd-moreno Chrsitina Sigurdson - Selective Cell Vulnerability: http://bit.ly/cjd-sigurdson Q&A with Deslys, Moreno, Sigurdson, and Knight: http://bit.ly/cjd-qa2 Emiliano Biasini - Pharmacological Chaperones http://bit.ly/cjd-biasini CJD Foundation Website: http://www.CJDFoundation.org Subscribe: https://goo.gl/hRMXAp -- "Prion diseases are a group of rare, invariably fatal brain disorders which occur both in humans and certain animals. They first came to public attention in the mid 1980s in the form of the BSE epidemic in the United Kingdom. BSE (bovine spongiform encephalopathy) is a prion disease in cattle. Tissue from infected animals may have contaminated cattle feed, leading to the silent spread of the BSE epidemic. There is also a theory that BSE came from feed contaminated with scrapie, the long established sheep prion disease. Inevitably, concern over whether BSE could pass to humans mounted. In humans the best known of the prion diseases is Creutzfeldt-Jakob Disease (CJD), which reportedly affects around one person per million per year. In the United States this translates to 250-300 new cases per year. It is well known that CJD is very difficult to diagnose leading to speculation that the one case per million report may be incorrect. Most of the cases are "classical" or "sporadic" CJD (sCJD), occurring for no, as yet, known reason. The sporadic form accounts for approximately 85% of the cases, the familial form approximately 15%. There have also been a few cases which have occurred from contamination via medical procedures; this type is known as iatrogenic or Acquired CJD. Finally over the last few years, another type of Acquired CJD called variant (vCJD) has been identified in young people. CJD has been linked to ingestion of beef tainted with BSE (bovine spongiform encephalopathy), most cases have occurred in the United Kingdom." -- Information Provided by: http://www.cjdfoundation.org/about-cjd
https://wn.com/Prion_Disease_And_Cjd_Vaccination_Approaches_By_Thomas_Wisniewski
Mad Cow Disease and Food Safety News Programme (Full length)

Mad Cow Disease and Food Safety News Programme (Full length)

  • Order:
  • Duration: 29:13
  • Updated: 01 Jul 2011
  • views: 16647
videos
See http://www.foodsafetypolicy.com/documentary for the interview towards the end of the programme. Joe Crowley's (the presenter's) filming diary... For me and friends of my age, Mad Cow Disease seems like a distant nightmare - something that haunted us on the news as we grew up. But this Inside Out programme has made me think again and, sadly, given the disease a painful new relevance. It's about a young guy like me, starting out on a career in the media, when he was struck down by the human form of the disease. And it's a story of his mother's (Christine Lord) relentless search for answers, as she tries to find out who killed her son (Andrew Black). *** Further informarion on variant CJD Inside Out looks at the rise of Variant CJD, the symptoms and the support available for those caring for or mourning the loss of loved ones from the disease. Variant CJD Variant CJD is a form of transmissible dementia and differs from other forms of CJD. It has received a lot of media attention over recent years, but has been around for much longer. Early 1920s CJD is first recognised by two German neurologists Creuzfeldt and Jakob, the namesakes of the disease. Following decades It is a rare disease with an estimated UK occurrence of 30-50 cases per year. Dec 1984 Cow number 133 on Stent farm Sussex becomes the first confirmed victim of BSE the animal strain of CJD. Feb 1985 Cow 133 dies. Nov 1986 BSE is first formally identified by the Central Veterinary Laboratory. April 1988 The Government establishes the Southwood committee to look into BSE. It concludes that BSE is probably spread in animal feed. May 1990 Chief Medical Officer says beef is safe to eat. July 1993 100,000th case of BSE in Britain. May 1995 First death from Variant CJD. March 1996 Government announces suspected link between BSE and the human equivalent CJD . The EU announces worldwide export ban on all British beef. March 1998 A public inquiry into the origin and spread of BSE and CJD opens in London. Jan 2000 Claire Mcvey, age 15, dies of Variant CJD. August 2002 92 deaths in Britain to date from definite Variant CJD . 22 deaths from probable Variant CJD. Symptoms Although symptoms are diverse across patients, a number of similarities have been found when sufferers' brain were examined under a microscope. According to the Crentzfelt-Jakob Support Network, most had lost nerve cells in the brain leading to a spongy appearance. There are many symptoms of Variant CJD, which can broadly be split into two categories... Physical symptoms: Unsteadiness on feet Development of uncontrollable jerky movements Progressive stiffness of the limbs Incontinence Mental symptoms: The progressive loss of mental abilities Mood swings Loss of awareness of surroundings The incubation period for the disease is years rather than weeks or months. Therefore, the symptoms get progressively worse over time. Medicine According to the Crentzfelt-Jakob Support Network, most people with CJD die within six months. But in about 10 per cent of cases it can last between two and five years. At present, there is no known cure for CJD despite considerable, ongoing research. There are some medicines which make the last few weeks easier for sufferers. Source of information: http://www.bbc.co.uk/insideout/southwest/series1/claire-mcvey.shtml
https://wn.com/Mad_Cow_Disease_And_Food_Safety_News_Programme_(Full_Length)
UK: LONDON: BSE CRISIS LATEST

UK: LONDON: BSE CRISIS LATEST

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  • Duration: 2:41
  • Updated: 21 Jul 2015
  • views: 362
videos
English/Nat XFA European farm ministers are meeting in Brussels on Monday for crisis talks expected to result in tough new restrictions on cattle feed to control the spread of mad cow disease throughout the continent. The restrictions, set to be imposed throughout the European Union, are more stringent than those introduced by Britain in 1996 following the establishment of a probably link between the disease, also known as BSE, and the lethal human condition variant CJD. The existing ban on meat and bonemeal (MBM) feed for cattle could be extended to all other animals including pigs, chickens and pets, and feed containing fish meal and chicken remains could also be banned. Monday's meeting comes after panic in France, where BSE cases are rising and four human victims of vCJD have been identified, and Germany, which has just reported its first infected cow. Britain's Agriculture Minister Nick Brown on Sunday said he was prepared to accept the proposed Europe-wide controls on beef production, which go further than British scientists have so far deemed necessary. He stressed that action had to be taken at an EU-wide level as unilateral national bans would not be effective. The feeding of MBM to cows has been blamed for spreading BSE. Its use for ruminants has been banned across the EU since 1994, but it had remained in use for pigs and poultry. The EU is not recommending a blockade on French beef exports, such as that imposed on Britain for more than three years when its first BSE outbreak was discovered. This has angered British beef traders, who feel that all their efforts to improve their industry will be undone if beef from affected countries is not banned. SOUNDBITE: "We know in the meat trade that there has been all sorts of beef coming in from possibly Germany and Holland especially and we understand now through this latest problem that French beef has been coming in through those channels as far as I'm concerned that should be totally banned. We've got the safest beef in the world now in this country. Why jeopardise that by bringing this other beef in?" SUPER CAPTION: John Absalom, Deputy Chairman, Smithfield Tenants' Association SOUNDBITE: "Until the abattoirs on the continent are doing the same sort of thing as England, if they can get the cow meat out of their trade which we've not had in since our regulations were introduced, then you're beginning to suffer again." SUPER CAPTION: Colin West, Smithfield meat trader SOUNDBITE: (English) "It occurs to me that the only people that carry out the directives put out by Brussels are the British. The other 14 nations look at the EU directives and say 'Oh yeh they're nice' and push them to one side, and we go on with it. Now we've decimated our cattle herds..." SUPER CAPTION: Dan Harmston, Self employed meat porter You can license this story through AP Archive: http://www.aparchive.com/metadata/youtube/3326d16bf9319e93a4e8f0242743c035 Find out more about AP Archive: http://www.aparchive.com/HowWeWork
https://wn.com/UK_London_Bse_Crisis_Latest
BSE symptom video

BSE symptom video

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  • Duration: 0:48
  • Updated: 02 May 2015
  • views: 15730
videos
https://wn.com/Bse_Symptom_Video
BSE - UK

BSE - UK

  • Order:
  • Duration: 20:41
  • Updated: 07 Nov 2007
  • views: 10663
videos
July 2005 The cost of the crisis over BSE, and its links to the human variant CJD were obvious to all in terms of cattle and livelihood. However, the question of the role that cost had to play before the crisis erupted is one that is much more contentious. Ron McCullagh illustrates both the devastating impact of the crisis, and the debates that still rage over the nature and treatment of the disease.
https://wn.com/Bse_UK
How, and when, people could get variant CJD?

How, and when, people could get variant CJD?

  • Order:
  • Duration: 1:51
  • Updated: 03 Aug 2010
  • views: 1340
videos
Click here http://www.foodsafetypolicy.com/howandwhen Bovine spongiform encephalopathy (BSE), commonly known as mad-cow disease, is a fatal neurodegenerative disease in cattle that causes a spongy degeneration in the brain and spinal cord. BSE has a long incubation period, about 30 months to 8 years, usually affecting adult cattle at a peak age onset of four to five years, all breeds being equally susceptible. In the United Kingdom, the country worst affected, more than 180,000 cattle have been infected and 4.4 million slaughtered during the eradication program. The disease may be most easily transmitted to human beings by eating food contaminated with the brain, spinal cord or digestive tract of infected carcasses. However, it should also be noted that the infectious agent, although most highly concentrated in nervous tissue, can be found in virtually all tissues throughout the body, including blood. In humans, it is known as new variant Creutzfeldt--Jakob disease (vCJD or nvCJD), and by October 2009, it had killed 166 people in the United Kingdom, and 44 elsewhere[5] Between 460,000 and 482,000 BSE-infected animals had entered the human food chain before controls on high-risk offal were introduced in 1989. A British inquiry into BSE concluded that the epizootic was caused by cattle, who are normally herbivores, being fed the remains of other cattle in the form of meat and bone meal (MBM), which caused the infectious agent to spread. There are studies indicating that the cause of BSE may be from the contamination of MBM from sheep with scrapie that were processed in the same slaughterhouse. The epidemic was probably accelerated by the recycling of infected bovine tissues prior to the recognition of BSE. The origin of the disease itself remains unknown. The infectious agent is distinctive for the high temperatures at which it remains viable; this contributed to the spread of the disease in the United Kingdom, which had reduced the temperatures used during its rendering process. Another contributory factor was the feeding of infected protein supplements to very young calves. This first reported case in North America was in December 1993 from Alberta, Canada. Another case reported later in May 2003. The first known U.S. occurrence came in December of the same year though it was later confirmed that it was a cow of Canadian origin and imported to the U.S. Canada announced two additional cases of BSE from Alberta in early 2005. In June 2005 Dr. John Clifford, chief veterinary officer for the United States Department of Agriculture animal health inspection service confirmed a fully domestic case of BSE in Texas. Dr. Clifford would not identify the ranch, calling that "privileged information." The 12 year old animal was alive at the time when Oprah Winfrey raised concerns about cannibalistic feeding practices on her show which aired April 16, 1996.
https://wn.com/How,_And_When,_People_Could_Get_Variant_Cjd
Theory on the origins of BSE (2005): United Kingdom video

Theory on the origins of BSE (2005): United Kingdom video

  • Order:
  • Duration: 9:57
  • Updated: 30 Dec 2008
  • views: 4148
videos
Click herehttp://www.foodsafetypolicy.com New variant CJD may have come from human remains which were put into cattle feed. This theory has been criticised but it is some interesting research into the causes of BSE. *** CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts. The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordination dysfunction (ataxia), changes in gait, rigid posture, and seizures. The duration of the disease varies greatly, but sporadic (non-inherited) CJD can be fatal within months or even weeks. In some people, the symptoms can continue for years. In most patients, these symptoms are followed by involuntary movements and the appearance of an atypical diagnostic electroencephalogram tracing. Most victims die 6 months after initial symptoms appear, often of pneumonia due to impaired coughing reflexes. About 15% of patients survive 2 or more years. The symptoms of CJD are caused by the progressive death of the brain's nerve cells, which is associated with the build-up of abnormal prion proteins forming amyloids. When brain tissue from a CJD patient is examined under a microscope, many tiny holes can be seen where whole areas of nerve cells have died. The word "spongiform" in "transmissible spongiform encephalopathies" refers to the sponge-like appearance of the brain tissue. Cause Transmissible spongiform encephalopathy diseases are caused by prions. The diseases are thus sometimes called prion diseases. Other prion diseases include Gerstmann--Sträussler--Scheinker syndrome (GSS), fatal familial insomnia (FFI) and kuru in humans, as well as bovine spongiform encephalopathy (BSE, commonly known as mad cow disease) in cattle, chronic wasting disease (CWD) in elk and deer, and scrapie in sheep. Alpers' syndrome in infants is also thought to be a transmissible spongiform encephalopathy caused by a prion.
https://wn.com/Theory_On_The_Origins_Of_Bse_(2005)_United_Kingdom_Video
UK: TEENAGER WHO DIED FROM HUMAN EQUIVALENT OF MAD COW DISEASE

UK: TEENAGER WHO DIED FROM HUMAN EQUIVALENT OF MAD COW DISEASE

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  • Duration: 1:45
  • Updated: 21 Jul 2015
  • views: 497
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English/Nat The parents of a teenager who died from the human equivalent of Mad Cow Disease hit out Thursday at the British government's handling of the issue. Stephen Churchill was 19 when he died last year from Disease. The government has only now admitted there's a link between the two conditions. Their grief dates from a year ago - but their anger is new. Dorothy and David Churchill say the death of their son Stephen had been a mystery - until now. They recall how his illness started. SOUNDBITE: (English) "All the things that you and I do naturally, take for granted, Stephen couldn't do. He couldn't feed himself, he couldn't walk, he had no coordination, his short term memory went, couldn't write and he actually was only in bed the last few days of his life. Prior to that he did get up and sit in a chair but he couldn't walk, he had to be moved in a wheelchair." SUPER CAPTION: Dorothy Churchill, Mother Stephen's father tells how initially the doctors were taken completely by surprise. SOUNDBITE: (English) "Well we were, perhaps even like the medics, we were very confused because when Stephen was in the psychiatric ward they said, well this is a severe depression but with bizarre symptoms. Then when he went to the general hospital it was diagnosed that it was a progressive, degenerative, neurotic disease but they didn't have a name for it. We now know the reasons why, that's because Stephen's age was confusing people. CJD didn't strike people that old it was always the disease of the 50, 60 or 70 year old." SUPER CAPTION: David Churchill, Father Stephen's parents are angry that only now has the UK government accepted there's a link between Mad Cow Disease and the illness he died from - Creutzfeldt- Jakob Disease - and that British beef could be to blame. SOUNDBITE: (English) "Shocked, angry, confused, you know if you'd asked me yesterday I'd say I really don't think there is anything in beef but now we're told Stephen is one of the 10 that was infected through BSE beef and it just makes you so angry." SUPER CAPTION: Dorothy, Stephen's mother Now they want an independent inquiry into the affair. You can license this story through AP Archive: http://www.aparchive.com/metadata/youtube/544050ffccdd8ab62fc60a0e8a5af3d6 Find out more about AP Archive: http://www.aparchive.com/HowWeWork
https://wn.com/UK_Teenager_Who_Died_From_Human_Equivalent_Of_Mad_Cow_Disease
Prion Disease | Gerstmann-Straussler Scheinker (GSS) Syndrome by Julia Moreno

Prion Disease | Gerstmann-Straussler Scheinker (GSS) Syndrome by Julia Moreno

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  • Duration: 18:12
  • Updated: 03 Sep 2015
  • views: 2084
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Discussing the specific Prion Disease, Gerstmann-Straussler Scheinker (GSS) Syndrome, Julia Moreno showcases her studies on the role of Cellular Chaperons, and the Unfolded Protein Response in the Gerstmann-Straussler Scheinker (GSS) Syndrome. 0:30 - Human Prion Disease Types 1:29 - Gerstmann-Straussier Scheinker (GSS) 2:30 - Spontaneous Neurodegenerative Disease in Tg(GSS)mice 4:15 - Mis-folds During Prion Disease 5:57 - Hypothesis 6:15 - Generation of Susceptible (S) and Resistant (R) Cells Study 7:46 - Identification of Differences between S and R Cells 9:07 - PANTHER Analysis 15:27 - Conclusions -- Watch more from this conference at: 2015 CJD Foundation Family Conference Playlist: http://bit.ly/cjd-2015 Individual Presentations: Byron Caughey - Detecting Prions: http://bit.ly/cjd-caughey Glenn Telling - Transgenic Mouse Modeling: http://bit.ly/cjd-telling Thomas Wisniewski - Vaccination Approaches: http://bit.ly/cjd-wisniewski Q&A with Caughey, Telling, Wisniewski, Will: http://bit.ly/cjd-qa1 Jean-Phillipe Deslys: Human Mini-Brains: http://bit.ly/cjd-deslys Julia Moreno: Gerstamann-Straussler Scheinker Syndrome: http://bit.ly/cjd-moreno Chrsitina Sigurdson - Selective Cell Vulnerability: http://bit.ly/cjd-sigurdson Q&A with Deslys, Moreno, Sigurdson, and Knight: http://bit.ly/cjd-qa2 Emiliano Biasini - Pharmacological Chaperones http://bit.ly/cjd-biasini CJD Foundation Website: http://www.CJDFoundation.org Subscribe: https://goo.gl/hRMXAp -- "Prion diseases are a group of rare, invariably fatal brain disorders which occur both in humans and certain animals. They first came to public attention in the mid 1980s in the form of the BSE epidemic in the United Kingdom. BSE (bovine spongiform encephalopathy) is a prion disease in cattle. Tissue from infected animals may have contaminated cattle feed, leading to the silent spread of the BSE epidemic. There is also a theory that BSE came from feed contaminated with scrapie, the long established sheep prion disease. Inevitably, concern over whether BSE could pass to humans mounted. In humans the best known of the prion diseases is Creutzfeldt-Jakob Disease (CJD), which reportedly affects around one person per million per year. In the United States this translates to 250-300 new cases per year. It is well known that CJD is very difficult to diagnose leading to speculation that the one case per million report may be incorrect. Most of the cases are "classical" or "sporadic" CJD (sCJD), occurring for no, as yet, known reason. The sporadic form accounts for approximately 85% of the cases, the familial form approximately 15%. There have also been a few cases which have occurred from contamination via medical procedures; this type is known as iatrogenic or Acquired CJD. Finally over the last few years, another type of Acquired CJD called variant (vCJD) has been identified in young people. CJD has been linked to ingestion of beef tainted with BSE (bovine spongiform encephalopathy), most cases have occurred in the United Kingdom." -- Information Provided by: http://www.cjdfoundation.org/about-cjd
https://wn.com/Prion_Disease_|_Gerstmann_Straussler_Scheinker_(Gss)_Syndrome_By_Julia_Moreno
Human 'Mad Cow Disease' (CJD) News Report No. 2

Human 'Mad Cow Disease' (CJD) News Report No. 2

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  • Duration: 3:28
  • Updated: 01 Oct 2013
  • views: 18550
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Click herehttp://www.foodsafetypolicy.com On December 2007, at just 24 years of age, Andy Black died after suffering from the human form of mad cow's disease vCJD (variant Creutzfeldt-Jakob Disease). Andy worked in the media producing/researching programmes for talkSPORT Radio, BBC, and ITV. During his terrible last days and at his request Christine Lord made it her mission to find out "Who killed my son?" and with the BBC, she produced a documentary exposing the key players at the centre of the BSE crisis whose actions and decisions led to his untimely and avoidable death. The Documentary is at http://www.youtube.com/watch?v=MAJTr6Nxxa0 Christine Lord, as Andy's mum and a freelance journalist continues to research the scandal - and its links between school meals, (state and independent), baby food, vaccines and medicines. *** Creutzfeldt--Jakob disease or CJD is a degenerative neurological disorder (brain disease) that is incurable and invariably fatal. CJD is at times called a human form of mad cow disease, given that bovine spongiform encephalopathy is believed to be the cause of variant Creutzfeldt--Jakob disease in humans. CJD is the most common among the types of transmissible spongiform encephalopathy found in humans. In CJD, the brain tissue develops holes and takes on a sponge-like texture. This is due to a type of infectious protein called a prion. Prions are misfolded proteins which replicate by converting their properly folded counterparts. The first symptom of CJD is rapidly progressive dementia, leading to memory loss, personality changes and hallucinations. This is accompanied by physical problems such as speech impairment, jerky movements (myoclonus), balance and coordination dysfunction (ataxia), changes in gait, rigid posture, and seizures. The duration of the disease varies greatly, but sporadic (non-inherited) CJD can be fatal within months or even weeks (Johnson, 1998). In some people, the symptoms can continue for years. In most patients, these symptoms are followed by involuntary movements and the appearance of an atypical diagnostic electroencephalogram tracing. Most victims die 6 months after initial symptoms appear, often of pneumonia due to impaired coughing reflexes. About 15% of patients survive 2 or more years. The symptoms of CJD are caused by the progressive death of the brain's nerve cells, which is associated with the build-up of abnormal prion proteins forming amyloids. When brain tissue from a CJD patient is examined under a microscope, many tiny holes can be seen where whole areas of nerve cells have died. The word "spongiform" in "transmissible spongiform encephalopathies" refers to the sponge-like appearance of the brain tissue.
https://wn.com/Human_'Mad_Cow_Disease'_(Cjd)_News_Report_No._2
Creutzfeldt-Jakob Disease (CJD) Q&A | Byron Caughey, Glenn Telling, Thomas Wisniewski, Robert Will

Creutzfeldt-Jakob Disease (CJD) Q&A | Byron Caughey, Glenn Telling, Thomas Wisniewski, Robert Will

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  • Duration: 37:33
  • Updated: 03 Sep 2015
  • views: 715
videos
During the Creutzfeldt-Jakob Disease (CJD) question and answer discussion board, leading experts on Prion Disease and similar disorders comment on what CJD is, discuss vaccination approaches to Prion Disease and talk about other aspects of Creutzfeldt-Jakob disease. Prion Disease and CJD Discussion Board: Byron Caughey, Glenn Telling, Thomas Wisniewski, and Robert Will 0:34 - Testing CJD Disease Results 1:31 - Creutzfeldt-Jakob Disease Test Turnout Time 5:00 - CJD Infection Analysis 8:42 - Pre-CJD Symptoms 14:11 - CWD Prevention 22:35 - CWD Immunity 27:24 - How to Detect CWD and Related Diseases 31:01 - Early Detection of CJD and Prion Disease 36:37 - Learning About CJD Through Similar Diseases -- Watch more from this conference at: 2015 CJD Foundation Family Conference Playlist: http://bit.ly/cjd-2015 Individual Presentations: Byron Caughey - Detecting Prions: http://bit.ly/cjd-caughey Glenn Telling - Transgenic Mouse Modeling: http://bit.ly/cjd-telling Thomas Wisniewski - Vaccination Approaches: http://bit.ly/cjd-wisniewski Q&A with Caughey, Telling, Wisniewski, Will: http://bit.ly/cjd-qa1 Jean-Phillipe Deslys: Human Mini-Brains: http://bit.ly/cjd-deslys Julia Moreno: Gerstamann-Straussler Scheinker Syndrome: http://bit.ly/cjd-moreno Chrsitina Sigurdson - Selective Cell Vulnerability: http://bit.ly/cjd-sigurdson Q&A with Deslys, Moreno, Sigurdson, and Knight: http://bit.ly/cjd-qa2 Emiliano Biasini - Pharmacological Chaperones http://bit.ly/cjd-biasini CJD Foundation Website: http://www.CJDFoundation.org Subscribe: https://goo.gl/hRMXAp -- "Prion diseases are a group of rare, invariably fatal brain disorders which occur both in humans and certain animals. They first came to public attention in the mid 1980s in the form of the BSE epidemic in the United Kingdom. BSE (bovine spongiform encephalopathy) is a prion disease in cattle. Tissue from infected animals may have contaminated cattle feed, leading to the silent spread of the BSE epidemic. There is also a theory that BSE came from feed contaminated with scrapie, the long established sheep prion disease. Inevitably, concern over whether BSE could pass to humans mounted. In humans the best known of the prion diseases is Creutzfeldt-Jakob Disease (CJD), which reportedly affects around one person per million per year. In the United States this translates to 250-300 new cases per year. It is well known that CJD is very difficult to diagnose leading to speculation that the one case per million report may be incorrect. Most of the cases are "classical" or "sporadic" CJD (sCJD), occurring for no, as yet, known reason. The sporadic form accounts for approximately 85% of the cases, the familial form approximately 15%. There have also been a few cases which have occurred from contamination via medical procedures; this type is known as iatrogenic or Acquired CJD. Finally over the last few years, another type of Acquired CJD called variant (vCJD) has been identified in young people. CJD has been linked to ingestion of beef tainted with BSE (bovine spongiform encephalopathy), most cases have occurred in the United Kingdom." -- Information Provided by: http://www.cjdfoundation.org/about-cjd
https://wn.com/Creutzfeldt_Jakob_Disease_(Cjd)_Q_A_|_Byron_Caughey,_Glenn_Telling,_Thomas_Wisniewski,_Robert_Will
Mad Cow Disease

Mad Cow Disease

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  • Duration: 2:19
  • Updated: 06 Mar 2015
  • views: 324665
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Also known as Creutzfeldt Jakob Disease in humans, it will attack your brain and body with deadly, incurable symptoms. Subscribe for new videos: http://goo.gl/SaufF4 Watch the scorpion video here: http://goo.gl/MYiSLm Watch the Brazilian wandering spider video here: http://goo.gl/Z6dFJS Infection from this disease always leads to death…and there is no cure. Today's video was produced due to high demand by our subscribers. If you have any other topics you'd like to learn about, be sure to write your suggestions below What is it? Mad cow disease, or bovine spongiform encephalopathy (BSE), is a disease that affects a cow's nervous system by putting holes in its brain, causing it to become spongy and start wasting away. The disease can also affect other tissues throughout the cow's body as well as its blood, making it easily transferrable to humans. When it's transmitted in humans, it's known as Creutzfeldt-Jakob Disease. Where is it located? The mad cow disease outbreak began in the United Kingdom, causing an international epidemic of the Creutzfeldt-Jakob Disease. As you can see here from the dark green areas, humans were infected throughout the UK and mainland Europe as well as all of North America and some countries in the east. The light green areas show countries in which only the cows were infected without affecting any humans. How will it kill you? Humans infected by mad cow disease meat begin experiencing symptoms of dementia, memory loss, and hallucinations followed by speech impairment, jerky movements, and seizures. Post-mortem observations of victims' brains have shown tiny holes in areas where their nerve cells have died. Death is progressive and eventually happens in about six months. How to Survive: There is no cure. Some patients have been known to live for about 4-5 years after infection, although their symptoms progress as they get closer to inevitable death. Governments have taken measures to prevent transmission from cattle to humans by killing millions of high-risk cows in an attempt to get rid of the disease. Also, the USDA now requires certain cow parts to be removed from high-risk cattle that show any signs of neurological problems to prevent them from entering the U.S. food supply. Now what do you think is worse and why? Being stung by a scorpion? Or being stung by a Brazilian wandering spider?
https://wn.com/Mad_Cow_Disease